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Consequently, MYD88 functions as an adapter protein for TLRs and, through its interaction with the intracellular domain of TLRs, may facilitate the recruitment of additional signaling molecules, ultimately resulting in the activation of the NF-B signaling pathway

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doi: 10.3389/fmicb.2020.579804 Received 03 July 2020 Accepted 15 October 2020 Published 12 November 2020 Volume 11 - 2020 Edited by Carlo Giuseppe Rizzello, University of Bari Aldo Moro, Italy Reviewed by Huaxi Yi, Ocean University of China, China

Two RCTs examined the efficacy of tanezumab vs